Pathophysiology Practice Test (17)

Pathophysiology Practice Test (17)

<<<<<<<<<<< Liver Insufficiency>>>>>>>>>>> ................. . Multiple Choice. ......................

published on June 21, 201214 responses 0

what may present in patients with hepatic insufficiency?

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encephalopathy
hypoproteinemia
hyperinsulinemia
renal failure

precipitating causes of hepatic encephalopathy is?

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gastrointestinal hemorrhage
alkalosis due to diuretic
constipation
infection

hyperammonemia in patient with cirrhosis related to?

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inadequate digestion of protein and excess
reproduction of intestinal bacteria
disorder of kidney
alkalosis
increased ammonia formation of the mucles

imbalance of plasma amino acid may lead to?

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less AAA enter the brain
more BCAA enter the brain
decreased formation of dopamine
increased formation of false neurotransmitter

hepatic encephalopathy lead by false neurotransmitter is because?

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FNT may compete with true neurotransmitter
disturb production of energy
FNT accumulated in the synapses of the reticular
structure
increase GABA

the pathogenesis of disorder of energy metabolic in brain result from hyperammonemia are?

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increased consume of α - ketoglutaric acid
increased consume of NADH
increased consume of ATP
increased of acetyle-CoA

the proper treatment of hepatic encephalopathy is?

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L- dopa
reduce oral intake of protein
sterilizing the bowel with antibiotics such as neomycin
acidification of the gut lumen by giving lactulose

plasma amino acid imbalance may lead to?

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increased production of phenyl-ethanolamine
increased of 5- HT
increased production octopamine
decreased production of dopamine

the pathogenesis of hepatic encephalopathy by false neurotransmitter is?

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easily enter blood-brain barrier
there is specific receptor of FNT in membraine of
neuron
FNT may uptake and store and release by neuron
FNT play weak physiological effects

precipitating factors of hepatic encephalopathy may be?

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prerenal azotemia
sedative and anesthetic agents
drink excessive alcohol
operation

the pathogenesis of hepatic ascites is?

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portal hypertension
decreased plasma colloidal osmotic pressure
decreased GFR
excessive aldosterone

the patient with hepatic encephalopathy may have?

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increased AAA ( aromatic amino acid) in plasma
decreased GABA in plasma
decreased BCAA ( branched chain amino acid) in
plasma
decreased phenylalanine in plasma

the pathogenesis of hepatic encephalopathy result from hyperammonemia are?

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increased consume of ATP
decrease production of ATP
increased formation of false neurotransmitter
increased formation of inhibited neurotransmitter

patients with hepatic cirrhosis may have?

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increased ADH in the body
increased ALD in the body
increased phagocytic role of kupffer
intestinal endotoxemia may occur cells

false neurotransmitter refers to?

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phenyl-ethanolamine
phenyl-ethylamine
octopamine
tyromine